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Lawrence Boise
Lawrence Boise
Verified email at emory.edu
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Year
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1
DJ Klionsky, AK Abdel-Aziz, S Abdelfatah, M Abdellatif, A Abdoli, S Abel, ...
autophagy 17 (1), 1-382, 2021
13238*2021
bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death
LH Boise, M González-García, CE Postema, L Ding, T Lindsten, LA Turka, ...
cell 74 (4), 597-608, 1993
41661993
Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death
E Yang, J Zha, J Jockel, LH Boise, CB Thompson, SJ Korsmeyer
Cell 80 (2), 285-291, 1995
28491995
CD28 costimulation can promote T cell survival by enhancing the expression of Bcl-XL
LH Boise, AJ Minn, PJ Noel, CH June, MA Accavitti, T Lindsten, ...
Immunity 3 (1), 87-98, 1995
16611995
Caspase-9, caspase-3 and caspase-7 have distinct roles during intrinsic apoptosis
M Brentnall, L Rodriguez-Menocal, RL De Guevara, E Cepero, LH Boise
BMC cell biology 14, 1-9, 2013
13052013
Proteasome inhibitors induce a terminal unfolded protein response in multiple myeloma cells
EA Obeng, LM Carlson, DM Gutman, WJ Harrington Jr, KP Lee, LH Boise
Blood 107 (12), 4907-4916, 2006
12832006
Multiple Bcl-2 family members demonstrate selective dimerizations with Bax.
TW Sedlak, ZN Oltvai, E Yang, K Wang, LH Boise, CB Thompson, ...
Proceedings of the National Academy of Sciences 92 (17), 7834-7838, 1995
11191995
Interactions among members of the Bcl-2 protein family analyzed with a yeast two-hybrid system.
T Sato, M Hanada, S Bodrug, S Irie, N Iwama, LH Boise, CB Thompson, ...
Proceedings of the National Academy of Sciences 91 (20), 9238-9242, 1994
8251994
Bax-independent inhibition of apoptosis by Bcl-XL
EHY Cheng, B Levine, LH Boise, CB Thompson, JM Hardwick
Nature 379 (6565), 554-556, 1996
6441996
bcl-xLx is the major bcl-x mRNA form expressed during murine development and its product localizes to mitochondria
M González-García, R Pérez-Ballestero, L Ding, L Duan, LH Boise, ...
Development 120 (10), 3033-3042, 1994
5731994
Expression of bcl-xL can confer a multidrug resistance phenotype
AJ Minn, CM Rudin, LH Boise, CB Thompson
Blood 86 (5), 1903-1910, 1995
5591995
Bcl-XL and Bcl-2 repress a common pathway of cell death.
DT Chao, GP Linette, LH Boise, LS White, CB Thompson, SJ Korsmeyer
The Journal of experimental medicine 182 (3), 821-828, 1995
5241995
Discovery of Mcl-1-specific inhibitor AZD5991 and preclinical activity in multiple myeloma and acute myeloid leukemia
AE Tron, MA Belmonte, A Adam, BM Aquila, LH Boise, E Chiarparin, ...
Nature communications 9 (1), 5341, 2018
4512018
Bortezomib inhibits PKR-like endoplasmic reticulum (ER) kinase and induces apoptosis via ER stress in human pancreatic cancer cells
ST Nawrocki, JS Carew, K Dunner Jr, LH Boise, PJ Chiao, P Huang, ...
Cancer research 65 (24), 11510-11519, 2005
3772005
Ascorbic acid enhances arsenic trioxide–induced cytotoxicity in multiple myeloma cells
JM Grad, NJ Bahlis, I Reis, MM Oshiro, WS Dalton, LH Boise
Blood, The Journal of the American Society of Hematology 98 (3), 805-813, 2001
3712001
Expression of Bcl-xL and loss of p53 can cooperate to overcome a cell cycle checkpoint induced by mitotic spindle damage.
AJ Minn, LH Boise, CB Thompson
Genes & development 10 (20), 2621-2631, 1996
3501996
CD28 costimulation prevents cell death during primary T cell activation.
PJ Noel, LH Boise, JM Green, CB Thompson
Journal of immunology (Baltimore, Md.: 1950) 157 (2), 636-642, 1996
3461996
Identification of immunosuppressant-induced apoptosis in a murine B-cell line and its prevention by bcl-x but not bcl-2.
AR Gottschalk, LH Boise, CB Thompson, J Quintans
Proceedings of the National Academy of Sciences 91 (15), 7350-7354, 1994
3221994
Regulation of Bcl-xL: a little bit of this and a little bit of STAT
JM Grad, XR Zeng, LH Boise
Current opinion in oncology 12 (6), 543-549, 2000
3052000
Bcl-xL can inhibit apoptosis in cells that have undergone Fas-induced protease activation
LH Boise, CB Thompson
Proceedings of the National Academy of Sciences 94 (8), 3759-3764, 1997
2891997
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